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JCI - Myelin/oligodendrocyte glycoprotein–deficient (MOG-deficient) mice reveal lack of immune tolerance to MOG in wild-type mice
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Gene expression in oligodendrocytes during remyelination reveals cholesterol homeostasis as a therapeutic target in multiple sclerosis | PNAS
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Myelin Gene Regulatory Factor Is a Critical Transcriptional Regulator Required for CNS Myelination - ScienceDirect
![Active induction of experimental autoimmune encephalomyelitis by MOG35-55 peptide immunization is associated with differential responses in separate compartments of the choroid plexus | Fluids and Barriers of the CNS | Full Text Active induction of experimental autoimmune encephalomyelitis by MOG35-55 peptide immunization is associated with differential responses in separate compartments of the choroid plexus | Fluids and Barriers of the CNS | Full Text](https://media.springernature.com/full/springer-static/image/art%3A10.1186%2F2045-8118-9-15/MediaObjects/12987_2012_Article_46_Fig3_HTML.jpg)
Active induction of experimental autoimmune encephalomyelitis by MOG35-55 peptide immunization is associated with differential responses in separate compartments of the choroid plexus | Fluids and Barriers of the CNS | Full Text
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Induction of antigen-specific tolerance by hepatic AAV immunotherapy regardless of T cell epitope usage or mouse strain background: Molecular Therapy - Methods & Clinical Development
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Gene Therapy-Induced Antigen-Specific Tregs Inhibit Neuro-inflammation and Reverse Disease in a Mouse Model of Multiple Sclerosis: Molecular Therapy
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Cloning mog-1. (A) Genetic map. mog-1 is located between one cloned... | Download Scientific Diagram
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Alternative isoforms of myelin/oligodendrocyte glycoprotein with variable cytoplasmic domains are expressed in human brain - Allamargot - 2007 - Journal of Neurochemistry - Wiley Online Library
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Frontiers | QTL Mapping for Agronomic and Adaptive Traits Confirmed Pleiotropic Effect of mog Gene in Black Gram [Vigna mungo (L.) Hepper]
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Amyloid-like Behavior of Site-Specifically Citrullinated Myelin Oligodendrocyte Protein (MOG) Peptide Fragments inside EBV-Infected B-Cells Influences Their Cytotoxicity and Autoimmunogenicity | Biochemistry
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Frontiers | Targeting transmembrane-domain-less MOG expression to platelets prevents disease development in experimental autoimmune encephalomyelitis
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